Written in English
|Statement||by Donald G. Stevens.|
|The Physical Object|
|Pagination||60 leaves, bound :|
|Number of Pages||60|
Significant mortality occurred among coho fingerlings exposed\ud to μg /liter copper and higher for 31 days. Most of the survivors\ud of these concentrations were unable to adapt to seawater and died\ud within the first three days of challenge. McKim and Benoit () found that yg/liter copper had a severe effect on survival of juvenile trout where slightly less than three months exposure resulted in 15% mortality in water with an alkalinity and hardness between 40 and 45 mg/liter The data from the coho fry bioassay indicate that yg/liter has a definite effect (mean% mortality) on survival of coho fry exposed to copper for 30 days. The sublethal effects of copper on the sensory physiology of juvenile coho salmon (Oncorhynchus kisutch) were vivo field potential recordings from the olfactory epithelium (electro‐olfactograms) were used to measure the impacts of copper on the responses of olfactory receptor neurons to natural odorants (L‐serine and taurocholic acid) and an odorant mixture Cited by: The sublethal effects of copper on the sensory physiology of juvenile coho salmon (Oncorhynchus kisutch) were vivo field potential recordings from the olfactory epithelium (electro‐olfactograms) were used to measure the impacts of copper on the responses of olfactory receptor neurons to natural odorants (L‐serine and taurocholic acid) and an odorant mixture .
From these studies, copper seem to have a detrimental effect on the immune response. In coho salmon, increasing levels of copper interfered with vaccination to Vibrio anguillarum, resulting in an increased mortality due to bacterial infection (Stevens, ), and an increased susceptibility to IHN and IPN viruses was observed in rainbow trout and other fish species exposed . Copper deficiency results in decreased humoral and cell-mediated, as well as nonspecific immune function. Impairment of immune function may be highly correlated with an increased incidence of infection and higher mortality rates observed in copper-deficient animals. Abstract. Sublethal exposure of juvenile coho salmon (Oncorhynchus kisutch) to Cr (as sodium dichromate) in freshwater decreased their disease resistance and serum agglutinin production againstVibrio ate disease resistance experiments, in which groups of salmon were exposed to or mg Cr/L for two weeks and then injected in subgroups with one of four tenfold . Growth, blood health, antioxidant status and immune response in juvenile yellow catfish a study reported that EE2 exposure have no significant effect on growth performance of juvenile coho salmon M. Crampe, A. Langston, P.J. GlynnModulatory effects of disease, stress, copper, TBT and vitamin E on the immune system of flatfish.
Among Pacific salmonids (Oncorhynchus spp.), copper‐induced olfactory impairment has previously been shown to disrupt behaviors reliant on a functioning sense of smell. For juvenile coho salmon (O. kisutch), this includes predator avoidance behaviors triggered . Although juvenile coho salmon (Oncorhynchus kisutch) movement patterns in streams and their relationship with body mass and growth have been examined in previous studies, most observations were limited to one season or one stream section. In this study, we monitored the movement of juvenile coho salmon throughout their period of residence in a. Collectively, examination of these data indicates that copper is broadly toxic to the salmon olfactory nervous system. Consequently, short-term inﬂuxes of copper to surface waters may interfere with olfactory-mediated behaviors that are critical for the survival. They exposed juvenile coho salmon to copper and pitted them against cutthroat trout, a common coho predator. Copper's Effect on Alaska's Salmon - .